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Ectopic expression of LYL1 in T cells alters the ratio of p105 to the mature p50 protein, resulting in the repression of NF B dependent gene activation by p50 p50 homodimers. The significant homology between LYL1 and TAL 1 may also be relevant, as TAL 1 activation is associated with a large number of human T ALLs. Further work is needed to assess the frequency of nfkb1 gene alterations in human leukemias and lymphomas, and to evaluate the physiological significance of p105 interaction with LYL1 for oncogenesis..

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